Meanings of ecm proteins in anglès

Collagen IV supported the serum-deprived cells better than the other ECM proteins.

Results: Cellular adhesion was similar on all ECM proteins and unaffected by EPO.

The ECM proteins in asthmatics were also compared with a normal control group.

Previous reports have shown changes in the expression of ECM proteins during local inflammatory responses.

Conclusion: The physiological ECM proteins are more supportive of primary podocytic cultures compared with collagen I.

The excessive synthesis of ECM proteins deteriorates hepatic architecture and results in liver fibrosis and cirrhosis.

Besides, EHEC showed a decreased adhesion capacity to ECM proteins in the presence of the bioprotective strain.

The ASM cells and ECM proteins are each able to influence the behaviour and characteristics of the other.

Expression of ECM proteins in lens capsule and lens epithelium was analyzed by immunohistochemistry and Western blot analysis.

Our results demonstrate that the CP and MP approaches more effectively remove cellular materials but differentially retain ECM proteins.

LPS induced EC inflammatory activation accompanied by expression of ECM proteins, increase in LOX activity, and activation of Rho signaling.

However, we now know that ECM proteins control many cellular events through their interaction with cell-surface receptors and cytoplasmic signaling pathways.

ECM proteins and receptors elicit diverse molecular signals that allow cell adaptation to environmental changes and regulate growth and cell motility.

ASM cells have the potential to play a key role in these processes through the production and release of ECM proteins.

Thus, Lasp-1 is a dynamic focal adhesion protein necessary for cell migration and survival in response to growth factors and ECM proteins.

A progressive accumulation of all ECM proteins investigated was noted in the pathological mesangial matrix at week 2 and later time points.