1
Right hindlimb function caused by the left-side lesion recovered within 6 days.
2
Tbx4 is known to exert essential functions in skeletal and muscular hindlimb development.
3
The mouse hindlimb ischemic model was established by ligating the right femoral artery.
4
We identified a robust hindlimb-specific interaction between Pitx1 and a putative hindlimb-specific enhancer.
5
Resting blood pressure was not altered by the 6 week hindlimb compression intervention.
6
A 1-year-old male mixed breed dog presented for the evaluation of progressive hindlimb paresis.
7
Results: Significant improvements in hindlimb tissue perfusion and perfusion kinetics were observed with both biomaterials.
8
Our experiment isolated a small group of hindlimb muscles and simulated exercise via tetanic contractions.
9
Importantly, BBB scores showed improved hindlimb motor functional recovery.
10
Delay in insulin action was virtually eliminated, indicated by immediate dose-dependent increments in hindlimb glucose uptake.
11
This clinical trial shows that G-CSF-based PB-MNC transplantation is a feasible treatment for the ischemic hindlimb.
12
However, the hindlimb has previously been viewed as the dominant propulsive organ in swimming freshwater turtles.
13
Histologic study of the gastrocnemius muscle demonstrated evidence of atrophy and fibrosis in the left hindlimb.
14
Finally, a positive correlation was found between locomotor functional scores and hindlimb muscle size following SCI.
15
The hindlimb digits often are uniform in length.
16
Arterial density was evaluated by microangiography and measurement of capillary and arteriolar density in hindlimb muscles.
