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Then a couple of uniformed officers enter, followed closely by an emt.
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However, EMT also contributes to the initiation and development of primary tumors.
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Told the EMT I knew my name, the date, when he asked.
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Josh met her gaze, but then the EMT stepped in the way.
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Here we review recent literature on non-EMT functions of EMT-controlling transcription factors.
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RBBP4 inhibition reduced cell invasion and migration via regulating proteins related to epithelial-mesenchymaltransition.
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Furthermore, markers of stem cells and epithelial-mesenchymaltransition were assessed.
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Such changes could be involved in the epithelial-mesenchymaltransition and development of proliferative vitreous retinopathy.
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These findings suggest that blockade of IL-6-induced epithelial-mesenchymaltransition is an antitumor mechanism of metformin.
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This coincided with a switch in phenotype that resembled an epithelial-mesenchymaltransition (EMT).
Usage of epithelial to mesenchymal transition in English
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Recent studies have associated epithelialtomesenchymaltransition with the development of chemoresistance.
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Additionally, there was significant upregulation of genes associated with epithelialtomesenchymaltransition.
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FAM3C is a secreted factor, which is involved in the epithelialtomesenchymaltransition.
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Increased Src activity promotes this process and inhibition of Src suppresses epithelialtomesenchymaltransition.
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Proteins that regulate cell proliferation, invasion, and epithelialtomesenchymaltransition are among the identified ADAM12-interacting proteins.
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Understanding the epithelialtomesenchymaltransition may explain the aggressive pattern of spread frequently observed in this disease.
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This review focuses on upregulation of Src in cancer as it relates to chemoresistance and epithelialtomesenchymaltransition.
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Recent evidence has demonstrated that epithelialtomesenchymaltransition (EMT) is associated with chemoresistance in pancreatic carcinoma cells.
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We find no evidence at the cellular or molecular level for epithelialtomesenchymaltransition of labeled cells into myofibroblasts.
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The epithelialtomesenchymaltransition (EMT) is a developmental process enabling epithelial cells to gain a migratory mesenchymal phenotype.
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Although multiple Src functions may contribute to metastasis, recently Src has been shown to play a role in epithelialtomesenchymaltransition.
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These cells appear to separate from the anterior epithelium and undergo a dramatic change that is reminiscent of the epithelialtomesenchymaltransition (EMT).
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Background: The microRNA-200 family participates in the maintenance of an epithelial phenotype and loss of its expression can result in epithelialtomesenchymaltransition (EMT).
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The epithelialtomesenchymaltransition (EMT) plays crucial roles in the formation of the body plan and in the differentiation of multiple tissues and organs.
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We further demonstrated that depletion of HEIRCC reduce the epithelialtomesenchymaltransition (EMT) program by regulating expression levels of EMT-associated markers in RCC cells.
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Genome-wide analysis of H3K79me2 distribution revealed that fibroblast-specific genes associated with the epithelialtomesenchymaltransition lose H3K79me2 in the initial phases of reprogramming.
Translations for epithelial to mesenchymal transition