1
Molecular clonality analysis presented a clonal T-cell receptor gamma gene rearrangement.
2
To explore this possibility, the 5' flank of the human IFN- gamma gene was analyzed.
3
In addition, gamma gene expression is detectable in the livers of newborn athymic mice.
4
One V gamma gene is rearranged and expressed prethymically.
5
The activation of gamma gene promoter does not depend on the presence of an HS2 enhancer.
6
No immunoglobulin and TCR- gamma gene rearrangements were detected.
7
Central to control of the IFN- gamma gene is the transcription factor T-bet, whose induction is Stat-1 dependent.
8
The mutation is located in the distal member of the duplicated CCAAT box of the gamma gene.
9
Monoclonal T-cell receptor gamma gene rearrangement was detected in all the cases of T-cell large granular lymphocyte leukemia.
10
The aim of this study was to evaluate the diagnostic contribution of TCR- gamma gene rearrangement analysed by PCR.
11
Substitution at -117 of the A gamma gene may inhibit the interaction resulting in an A gamma HPFH phenotype.
12
EKLF does not activate the gamma gene, the CACCC sequence of which differs from that of the beta gene.
13
We used this system to investigate the mechanism whereby human HPFH mutations result in gamma gene expression in the adult.
14
Factors binding to this element in the IFN- gamma gene were present in extracts from resting and activated Jurkat T cells.
15
Nucleotide sequences of the membrane exons of human immunoglobulin pseudo- gamma gene and gamma 1 chain cDNA were determined and compared.
16
In contrast, CTL from mice with targeted disruption of the IFN- gamma gene are unable to clear virus from persistently infected mice.
