Meanings of myocardial infarct in English

Background: Inhibition of leukocyte adhesion can reduce myocardial infarct size in animals.

Significance: These findings may provide new insights in myocardial infarct healing and repair.

Thrombolytic therapy has been documented to reduce acute myocardial infarct size.

Similar results were obtained for incidence rates of myocardial infarct.

In contrast, propranolol and diltiazem did not show such protective effects on myocardial infarct size.

The acute administration of atorvastatin has been reported to reduce myocardial infarct size in animal studies.

These elements may be potentials tools to improve cardiac function in a swine myocardial infarct model.

In 12 sheep, infarct deformation was prevented by Marlex mesh placed over the anticipated myocardial infarct.

Results: Both ischemic and remifentanil post-conditioning reduced the myocardial infarct size relative to the control group in both models.

Also, the usefulness of image analysis as an objective method for the measurement of myocardial infarct area is evaluated.

In the present study we investigated the effect of short-term estrogen on myocardial infarct size in oophorectomized female rabbits.

Conclusions: The myocardial infarct size-limiting effect of preconditioning did not correlate with the degree of myocardial stunning accompanying preconditioning.

IPC displayed cardioprotection as evidenced by improved post-ischemic contractile recovery, decreased myocardial infarct size and reduced number of apoptotic cardiomyocytes.

The goal of this study was to compare the effects of these techniques in an experimental model of myocardial infarct.

We previously demonstrated that several epoxyeicosatrienoic acids (EETs) produce reductions in myocardial infarct size in rats and dogs.

The primary end point was coronary flow at the end of PCI procedure, and secondary end points included myocardial infarct size.