Significados de immune homeostasis en inglés

Mesenchymal stem cells have the capacity to maintain immune homeostasis and prevent autoimmunity.

Tregs prevent autoimmune disease, maintain immune homeostasis and modulate protective responses against infection.

Disassembly and phagocytic removal of dying cells is critical to maintain immune homeostasis.

Regulatory T (Treg) cells are important in maintaining self-tolerance and immune homeostasis.

Our results may emphasize the importance of retroelement-containing transcripts in maintaining host immune homeostasis.

Human regulatory T (T reg) cells are essential for immune homeostasis.

In addition, B-cells can also maintain immune homeostasis by expressing PD-L1 and suppressing T-cell activity.

The maintenance of immune homeostasis requires regulatory T cells (Treg cells).

Regulatory T cells (Tregs) maintain immune homeostasis by limiting inflammatory responses.

By controlling DC activation, cytokines regulate immune homeostasis and the balance between tolerance and immunity.

Thus, the impact of viral clearance on restoring immune homeostasis is being investigated by many groups.

Regulatory T cells (Treg) typically restrain overt inflammatory responses and maintain intestinal immune homeostasis.

Intestinal DCs orchestrate gut immune homeostasis by dampening proinflammatory T-cell responses and inducing anti-inflammatory IgA responses.

Yet, the interaction of colonizing bacterial organisms with the developing human intestine favors inflammation over immune homeostasis.

Accumulating evidence on the immunomodulatory roles of dsRNAs warrants a reconsideration of various models of immune homeostasis.

MΦ comprise a heterogeneous population of cells, which contribute to host defense and maintenance of immune homeostasis.