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1
Conclusions: RH and the development of new lesions may predict survival in
metastatic
ccRCC
.
2
We demonstrated that MYOF influences cellular proliferation of the
metastatic
CCRCC
cell line by regulating VEGFR2 degradation.
3
Combined therapies targeting the MYOF and VEGFR2 pathways might be effective against
metastatic
CCRCC
to increase patient survival.
4
Here we establish a molecular link between HIF stabilization and induction of AXL receptor expression in
metastatic
ccRCC
.
5
The expression of S100A6 was up-regulated in
metastatic
ccRCC
cells.
6
Importantly, genetic and therapeutic inactivation of AXL signaling in
metastatic
ccRCC
cells reversed the invasive and metastatic phenotype in vivo.
metastatic
ccrcc
metastatic