1However, information about the catabolism of heme in erythroid cells is limited.
2We conclude that normal Ikaros function increases normal apoptosis in erythroid cells.
3Bone marrow is generally considered the main source of erythroid cells.
4This receptor is expressed by subsets of endothelial, as well as erythroid cells.
5Finally, sustained expression of alternative FPN1 transcripts is apparently observed only in erythroid cells.
6This phenotype clearly differs from that induced by tyrosine kinase oncogenes in erythroid cells.
7Expression of globin genes in developing erythroid cells is controlled by upstream locus control regions.
8FOG-1 suppressed the proliferation of primitive and definitive erythroid cells in all stages of differentiation.
9We have previously shown that DMT1 deficiency impairs erythroid differentiation and induces apoptosis of erythroid cells.
10The aim of this study was to investigate whether erythroid cells possess specific mechanisms for iron export.
11In 16 younger adults and children, erythroid cells and platelets were predominantly derived from normal stem cells.
12Nucleated erythroid cells had high expression of ACKR1, which facilitated their direct contact with hematopoietic stem cells.
13Our results indicate that chGfi expression is restricted to erythroid cells of the primitive and definitive lineage.
14The underlying mechanisms for EKLF specifically restricted to erythroid cells are of great interest but remain incompletely understood.
15In our test system, the gamma-globin gene is expressed at similar levels in the embryonic and adult erythroid cells.
16When mouse bone marrow cells were induced with erythropoietin to differentiate into erythroid cells, the synthesis of bilirubin increased.
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