1
The authors suggest that acute fornix infarction may cause anterograde memory deficits.
2
Methods: Retrograde and anterograde tracing techniques combined with double-labeling immunofluorescence technique were used.
3
We also examined whether transneuronal viral transport in this model is exclusively anterograde.
4
In general, most proteins are transported independently in the anterograde direction.
5
The model was found to favor anterograde spreading of the aggregates.
6
Criteria of efficacy were the degree of sedation and anterograde amnesia.
7
Thus, the presence of amnesia would isolate anterograde contributions as the source of loss.
8
These results suggest that H.M.'s lexical word-retrieval skills remain fluid despite his profound anterograde amnesia.
9
Recent discoveries reveal a Golgi-centric spectrin-ankyrin skeleton required for Golgi integrity and anterograde protein trafficking.
10
Overall, recall of biographical information was positively correlated with clinical measures of anterograde episodic memory.
11
Electrophysiologic and anterograde tract tracing studies have demonstrated that the vagus nerve innervates the duodenum.
12
Kinesin is believed to generate force for the movement of organelles in anterograde axonal transport.
13
In anterograde amnesia, memory loss is obtained for events that occur subsequent to the traumatic insult.
14
Kinesin appears not to be required for the anterograde transport of synaptic vesicles or their components.
15
Additionally, we provide anterograde tracing results for the mouse brain, which corroborate the probabilistic tractography findings.
16
These data provide an empirical example of an anterograde-induced memory deficit that is independent of retrograde influences.
