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Purpose: To review critically the literature on pulmonary barotrauma in mechanically ventilated patients.
2
Transmurality was achieved in most lesions with no evidence of charring or barotrauma.
3
The mechanism held responsible is coagulation by the electrode of neighboring tissue and barotrauma.
4
Risk factors for barotrauma include high peak airway pressures, large tidal volumes and acute lung injury.
5
Observational clinical studies demonstrate an increased risk of barotrauma in the setting of acute lung injury.
6
Results: Lung histology confirmed pulmonary barotrauma and inflammation.
7
Articles investigating the aetiology or prevention of pulmonary barotrauma were critically evaluated according to published guidelines.
8
Conclusions: Rotational atherectomy causes atheroablation with only moderate evidence of barotrauma in heavily calcified arteries, even after adjunct balloon angioplasty.
9
Conclusion: NMBA improves oxygenation only after 48 h in moderate, severe ARDS patients and has a lower barotrauma risk without affecting ICU weakness.
10
Cisatracurium had a significantly lower risk of barotrauma than the control group with no difference in intensive care unit (ICU)-induced weakness.
11
This may rupture lung tissue (pulmonary barotrauma), which can lead to gas bubbles being released into the arterial circulation (arterial gas embolism).
12
There was no incidence of barotraumas.
13
Purpose: To review critically the literature on pulmonary barotrauma in mechanically ventilated patients.
14
Transmurality was achieved in most lesions with no evidence of charring or barotrauma.
15
The mechanism held responsible is coagulation by the electrode of neighboring tissue and barotrauma.
16
Risk factors for barotrauma include high peak airway pressures, large tidal volumes and acute lung injury.