Attack aircraft model by Ilyushin; sans suffixe version of Il-2 family.
1 In addition, EMFs increased the expression of IL-2 by the T cells.
2 Coincidentally, a deletion of this region enhanced the IL-2-induced expression of beta-casein.
3 The decreasing tolerance to IL-2 was associated primarily with elevations in creatinine.
4 Finally, despite impaired Rel-A translocation, T cells were capable of transcribing IL-2.
5 A decreased ability to secrete IL-2 suggests an element of infection-associated immunosuppression.
6 Preexisting antitumor precursors did not decrease when peptide plus IL-2 was administered.
7 Thyroid function should be measured routinely in cancer patients receiving IL-2-based treatment.
8 However, the relation between IL-2 and DC system needs to be further understood.
9 IL-2 was thought to be the causative agent in most cases of fever.
10 Low-dose IL-2 therapy has produced disappointing clinical response rates in melanoma.
11 Serum levels of sIL-2Ra and sTNF-RII were increased in volunteers who developed illness.
12 This study identified subsets of patients who tolerated less IL-2 with repeated courses.
13 Infusion of the cells was safe whether administered alone or with IL-2 subcutaneously.
14 This protection was not reversible by systemic administration of high doses of IL-2.
15 Similar inhibition of IL-2-induced proliferation was observed in the lungs, kidneys, and spleen.
16 The two IL-2 dose groups were combined for the primary analysis.
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