The resulting elevated triacylglycerol levels provide a likely explanation for accelerated atherosclerosis.

The triacylglycerol lipase food enzyme is intended to be used in baking processes.

Tissue POP concentrations correlated positively with percentage triacylglycerol and negatively with polar lipids.

Lipolysis of endogenous triacylglycerol was increased during the pre-ischaemic period in the fasted group.

However, the effects on triacyleride or triacylglycerol metabolism are complex and not fully clarified yet.

Moreover, circulating miR-130b was a better predictor of the metabolic syndrome than was triacylglycerol level.

Thus the present study suggests an association between PChE activity and the rate of triacylglycerol synthesis.

Tissue lipids were largely characterized by triacylglycerol, reflecting large energy reserves accumulated prior to reproductive migration.

These results were reflected in downregulation of late adipogenic factors, resulting in a decrease in triacylglycerol content.

The significance of these findings for the regulation of triacylglycerol synthesis under conditions in vivo is discussed.

These observations place lipin 1 as a potentially important link between triacylglycerol synthesis and adipose tissue inflammation.

There was almost no overlap in the genomic regions explaining variance between the triacylglycerol and phospholipid fractions.

Stress triggers triacylglycerol accumulation as lipid droplets.

This hypermetabolism may be an adaptive response to an inability to store triacylglycerol fuel in a normal manner.

Most of the label was incorporated into triacylglycerol fatty acids in normal as well as Zellweger's syndrome cells.

Windows that explained the largest proportions of genetic variance for individual lipids were found in the triacylglycerol fraction.