Purpose: Luminal breast cancer has a long natural history, with recurrences continuing beyond 10 years after diagnosis.
2
Conclusion: Luminal dimensions and plaque compositional features identified by OCT are minimally affected by observer variability, permitting dependable plaque classification.
3
Positive remodeling was also noted, which increased luminal area relative to control.
4
Background and purpose: Arterial remodeling may enable atherosclerotic disease without luminal stenosis.
5
The PFC response depended upon the number of DV-infected M luminal diameter.
6
Interstitial-to-luminal albumin clearance reached a maximum rate at 725 microM interstitial albumin.
7
These cells might arise from the luminal epithelium of the papillary duct.
8
Pathological examination revealed systemic vasculopathy with luminal narrowing and multi-laminated basement membranes.
9
Giant M phi may damage tubular epithelial cells from the luminal side.
10
At the luminal borders the cells were often irregular and displayed apocrine-like activity.
11
Thirty-three per cent of late luminal loss was due to chronic stent recoil.
12
Changes in luminal cross-sectional area in carbachol-contracted airways were measured using video endoscopy.
13
However, the influence of luminal pancreatic enzymes is not fully understood.
14
Vessels were then perfusion-fixed for morphometric analysis of luminal cross-sectional area.
15
Results: Basal-like lesions presented worse cancer-specific and disease-free survival compared to luminal tumors.
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Finally, analysis of phenotypic markers reveals heterogeneity within the luminal progenitor cell pool.