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Mechanism-based chemical kinetic models are increasingly being used to describe biologicalsignaling.
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Background: The ubiquity of protein-protein interactions in biologicalsignaling offers ample opportunities for therapeutic intervention.
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Consequently, biologicalsignaling and genetic networks not only translate stimuli with functional response but also random fluctuations.
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Our analysis refines the windows during which many key biologicalsignaling processes act in regeneration, including embryonic patterning signals, immune responses, bioelectrical signaling and apoptosis.
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Biologicalsignaling networks process extracellular cues to control important cell decisions such as death-survival, growth-quiescence, and proliferation-differentiation.