Significats de cellular excitability en anglès

K(ATP) channels couple intermediary metabolism to cellular excitability.

Such regulation allows a control of cellular excitability in conditions when pH is low but ATP concentration is normal.

Adrenal glucocorticoids exert powerful effects on cellular excitability in neuroendocrine cells and neurons, although the underlying mechanisms are poorly understood.

In recent years, a functional role for TASK channels in cellular excitability and vulnerability to anesthetics of motor neurons has been described.

Cross-modulation of Na(V) and K(V) channels by Na(V)β1 may promote diversity and flexibility in the overall control of cellular excitability and signaling.

ATP-sensitive K(+) channels are gated by intracellular ATP, allowing them to couple intermediary metabolism to cellular excitability, whereas the gating mechanism remains unclear.

Alternative exon splicing and reversible protein phosphorylation of large conductance calcium-activated potassium (BK) channels represent fundamental control mechanisms for the regulation of cellular excitability.