Conclusion: The classification criteria allow identifying centralsensitization pain following cancer treatment.
2
Capsaicin was applied on both forearms of ten healthy volunteers to induce peripheral and centralsensitization.
3
Animal studies indicate that glutamate plays a key role in pain transmission, centralsensitization, and cortical spreading depression.
4
The N-methyl-D-aspartate receptor (NMDAR) contributes to centralsensitization in the spinal cord and the generation of pain hypersensitivity.
5
The NMDAR-mediated astrocyte-to-neuron signalling thus represents a novel pathway that may contribute to the control of centralsensitization in pathological pain.
6
FGIDs in adults have been distinguished by enhanced responses of the central nervous system to pain stimuli, known as centralsensitization.
7
Chronicity and recurrence in musculoskeletal shoulder pain are highly prevalent and can possibly be attributed to the concept of centralsensitization.
8
Background: Attenuation of centralsensitization with NMDA-active drugs such as S-Ketamine may play a role in postoperative analgesia and prevention of neuropathic pain.
9
This suggests that CCI-ION induces early peripheral sensitization followed by development of centralsensitization that mediates spontaneous pain and contributes to mechanical hypersensitivity.
10
The identification and treatment of these pain generators lessens the total pain burden, facilitates rehabilitation and decreases the stimuli for ongoing centralsensitization.
11
This is indicative of neuroprotective action of S. platensis against glutamate excitotoxicity-induced centralsensitization produced by the peripheral joint inflammation in the collagen-induced arthritis.
12
Fibromyalgia is a widespread chronic pain disorder that is characterized in part by centralsensitization and increased pain response to peripheral nociceptive and non-nociceptive stimuli.
13
Recently, the CentralSensitization Inventory (CSI) was developed to assess symptoms of CS and central sensitivity syndromes.
14
Centralsensitization (CS) involves the amplification of neural signaling within the central nervous system, which evokes pain hypersensitivity.
15
Objective: Centralsensitization (CS), a mechanism explaining the persistence of symptoms, has been the focus of many research projects.
16
Objectives: Centralsensitization (CS) implies increased sensitivity of the nervous system, resulting in increased pain sensitivity as well as widespread pain.