Significats de chronic hf en anglès

Patients: A convenience sample of 66 patients with chronic HF were studied.

However, their long-term effects on outcomes in chronic HF are unknown.

Background: Vascular remodeling is a crucial compensatory mechanism in chronic HF.

Methods: Chronic HF was produced in 12 dogs by multiple sequential intracoronary microembolizations.

Conclusion: The use of potassium supplements in chronic HF was not associated with mortality.

Myogenic regulatory factors and NF-kB do not modulate diaphragm MyHC distribution during chronic HF.

Background: The prediction of SCD remains an important challenge in patients with mild-to-moderate chronic HF.

Conclusion: In well characterized patients with chronic HF, circulating miR-132 levels rise with the severity of HF.

Background: Mortality in chronic HF remains high, with a significant number of patients dying of progressive disease.

Conclusions: Intake of PUFAs and SFAs was independently associated with 1-y all-cause mortality in patients with chronic HF.

Although these findings are novel, they will need replication and validation in larger cohorts of chronic HF patients.

Methods and results: A sample of 280 adults with chronic HF were enrolled into a prospective cohort comparison study.

These findings provide compelling rationale for exploring the use of CCM for the treatment of patients with chronic HF.

We sought to investigate whether pyridostigmine would effectively reduce HR in patients with chronic HF as compared with ivabradine.

Conclusions: Elevated sST2 concentrations are predictive of SCD in patients with chronic HF and provide complementary information to NT-proBNP levels.

Methods: A secondary analysis of 203 patients with chronic HF enrolled in a randomized trial of sodium reduction was completed.