The cntn1 null mouse presents with ataxia, progressive muscle weakness, and postnatal lethality, similar to the affected members in this family.

However, little is known about the impact of CNT on hMSC behavior.

These investigations have demonstrated that some f-CNTs can directly elicitate specific inflammatory pathways.

It is up to the CNT to decide what happens now.

The first repeated in vitro stimulation of hippocampal neurons with CNT electrodes is demonstrated.