Corticotropin releasing factor and adrenocorticotropin stimulation tests, as well as 24-hour urinary-free cortisol measurements were performed.
2
When the cells are stimulated by the secretagogue 8-bromo-cAMP, these polypeptides are coordinately secreted with mature adrenocorticotropin into the culture medium.
3
In neither case was the sorting or secretion of adrenocorticotropin detectably altered, suggesting that the proteoglycan is not required for these processes.
4
The AtT-20 cell, a mouse pituitary tumor line that secretes adrenocorticotropin and beta-endorphin, sorts the proteins it externalizes into two exocytotic pathways.
5
Subsequently, 50% of the birds from each dosage group were subjected to a 6-day adrenal function test, whereby they were injected with long-lasting adrenocorticotropin (ACTH).
1
The effects of electrical stimulation of medial amygdala on the adrenocortical sensitivity to adrenocorticotrophin (ACTH) were investigated in hypophysectomized rats.
2
Subsequently, large fluctuations occurred in the adrenocorticotrophin concentration in the plasma of foetal sheep which were consistent with the stimulation of adrenocorticotrophin secretion.
1
The cardiovascular, fluid, electrolyte and endocrine responses to both steroid treatments were similar to those with corticotrophin.
2
The stress peptide corticotrophin-releasing factor (CRF) contributes to relapse to drug and alcohol seeking following withdrawal, although the cellular actions are poorly understood.
1
Plasma cortisol and adrenocorticotropichormone responses were similar to those found to experimentally-induced pain in normal subjects, i.e.
2
Plasma adrenocorticotropichormone levels were significantly higher in the CRF patients, possibly indicative of an underlying hypothalamic-pituitary-adrenal axis abnormality.
3
The serum cortisol and adrenal steroids levels were always nearly undetectable, but the adrenocorticotropichormone levels were extremely high.
4
However, blood pressure, mean heart rate and the plasma adrenocorticotropichormone level showed no significant differences between the two groups.
5
We studied four patients with adrenocorticotropichormone (ACTH)-independent hypercortisolism due to bilateral massive enlargement of the adrenal glands.
1
In all cases, cortisol level increased three-fold after adrenocorticotrophichormone.
2
Plasma adrenocorticotrophichormone (ACTH) and cortisol levels were determined at serial time points.
3
These sb mice also had significantly greater increases in plasma adrenocorticotrophichormone than mice with other genotypes after a stressful stimulus.
4
Low cortisol levels are almost universal in infants after treatment with dexamethasone, but the adrenal gland remains responsive to adrenocorticotrophichormone stimulation.
5
This study was designed to investigate the effect of chronic exercise on exercise-induced changes in plasma concentrations of adrenocorticotrophichormone (ACTH) and corticosterone in rats.
1
The best imaging approach for localizing ectopic ACTH-secreting tumors is not defined.
2
The rise in pressure was significant after 8 h of ACTH infusion.
3
The increase in cortisol levels was preceded by a corresponding ACTH increase.
4
The transport mechanism for precursor ACTH thus appears to recognize other prohormones.
5
Serum cortisol concentrations and ACTH stimulation tests were used to detect CIRCI.
Ús de corticotropin en anglès
1
These infusion rates produce plasma levels similar to those seen during corticotropin stimulation.
2
For example, corticotropin-releasing factor regulates the homeostatic pattern of visceral functions during stress.
3
Concentrations of endorphins and corticotropin increased significantly also during the anaerobic exercise test.
4
A Sheehan event probably occurred 33 years before the patient presented with corticotropin deficiency.
5
Thirty-one patients underwent also a human corticotropin releasing hormone test.
6
As with corticotropin there were only minor changes in the coronary and iliac vascular beds.
7
A rate-sensitive fast-feedback inhibition of stress-induced corticotropin secretion by glucocorticoids is well documented in rats.
8
By contrast, Thy1-alphaSyn mice had an increased fecal response to novelty stress and corticotropin releasing factor injected intraperipherally.
9
Three of seven cases contained cells reactive for ACTH and corticotropin-like intermediate lobe peptide CLIP or their precursor, pro-opiomelanocortin.
10
This blunted neuronal response was also associated with an attenuated stimulation of neuroendocrine corticotropin-releasing factor transcription and plasma corticosterone release.
11
Objective: To describe the course of autoimmune hyperthyroid disease in a patient with corticotropin (ACTH) deficiency treated with glucocorticoids.
12
These findings suggest that disruption of NO synthesis does not play a role in the development of corticotropin hypertension in sheep.
13
Peak cortisol responses to corticotropin releasing factor and adrenocorticotropic hormone were correlated with each other and with 24-hour urinary-free cortisol excretion.
14
The activation of brain corticotropin-releasing factor (CRF) pathways has a key role in the behavioral and visceral responses to stress.
15
Objective: To investigate whether genetic variants of the corticotropin-releasing factor system or their interaction influence the risk of developing AUD in chronic disease populations.
16
The possibility that corticotropin (ACTH)-induced hypertension results from a direct central effect of the adrenocortical steroids released by ACTH was investigated in sheep.