1
Previous studies on c-Abl- deficient mice have shown high post-natal mortality and lymphopenia.
2
Similar results were obtained in a genetic cross with LDL receptor- deficient mice.
3
We conclude that D2R- deficient mice are not a model of Parkinson's disease.
4
The plasma levels of Pi are similar in wild-type and NaPi-IIb- deficient mice.
5
Development of T and B cells in the CD28- deficient mice appeared normal.
6
TRAF6- deficient mice showed a severe defect in the Treg development in thymus.
7
IP receptor involvement was also demonstrated using TRPV1- deficient mice and IP-deficient mice.
8
Interestingly, Cobl- deficient mice furthermore showed a significant widening of the terminal web.
9
Renal adenosine levels were lower before and after ischemia in CD73- deficient mice.
10
T cells from Itch- and Cbl-b- deficient mice were resistant to anergy induction.
11
T cells from Itch- and CblB- deficient mice are resistant to anergy induction.
12
Acute tubular necrosis and neutrophil infiltration were markedly reduced in PKC-ε- deficient mice.
13
TIGIT-Fc inhibited delayed-type hypersensitivity reactions in wild-type but not interleukin 10- deficient mice.
14
The cultured teeth phenocopy the extra incisors phenotype of the Sostdc1- deficient mice.
15
Research design and methods: Reversa mice are LDL receptor- deficient mice that develop atherosclerosis.
16
We obtained similar findings using pancreatic islets prepared from RGS4- deficient mice.
