Survival of odontoblasts during infection and inflammation determines prognosis of the dentalpulp.
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The DPSCs were collected from the dentalpulp of adult human third molars.
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Inflammation and elevated IL17A secretion occurred in the dentalpulp during orthodontic tooth movement.
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Results: The active fission activity of mitochondria and mitophagy in inflammatory dentalpulp was observed.
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Pulpitis, inflammation of the dentalpulp, is a disease that often necessitates emergency dental care.
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Introduction: Chronic inflammation disrupts dentalpulp regeneration by disintegrating the recruitment process of progenitors for repair.
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Thus, our data suggest that stem cells derived from dentalpulp may have therapeutic benefits for PD.
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Stable cell lines of dentalpulp stem cells are generated, maintained in culture, and used for subsequent experiments.
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The treatment enhanced dentalpulp cell adhesion and proliferation, suggesting that this system is promising for medical applications.
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We identified activation of autophagy and apoptosis in the odontoblasts due to inflammation of the human dentalpulp.
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Information concerning cell proliferation and differentiation in dentalpulp may be important to understand tooth response to exogenous stimuli.
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A spraying method for building-up polyelectrolyte films followed by the deposition of dentalpulp cells by spraying is presented.
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In this study, we investigated the mitochondrial damage and mitophagy occurred in inflammatory human dentalpulp and lipopolysaccharide-stimulated preodontoblasts.
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Furthermore, adult dentalpulp cells supplemented with these two growth factors re-established the inductive potential of postnatal dentalpulp tissue.
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Immunohistochemical staining of infected human dentalpulp and tissue from experimental dental pulpitis in rats showed that Wnt5a levels were increased.
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Up-regulation of VEGF expression in the dentalpulp may result in increased intra-pulpal pressure, and contribute to pain and irreversible tissue damage.