Significats de dependent relaxation en anglès

Endothelium- dependent relaxation of aortic rings was impaired in mice exposed to stress.

It has been reported that PTX induces endothelium- dependent relaxation of vascular smooth muscle.

Endothelium- dependent relaxation was almost completely abrogated in cholesterol-fed rabbits.

Although depressed endothelium- dependent relaxation occurs during early sepsis, the precise mechanism responsible for this remains unknown.

In contrast, CGRP, substance P, and VIP all produced a concentration- dependent relaxation of both arterial regions.

Endothelium- dependent relaxation was intact in the control and proximal sites and impaired in the stenotic sites.

Chronic exposure to cocaine significantly decreases beta-adrenoceptor-mediated relaxation and blunts endothelium- dependent relaxation to a small degree.

Angioplasty caused ruptures and dissections and abolished tone that returned after 5 days of perfusion along with BK- dependent relaxation.

In the present study we have analyzed the effects of short-term arsenite exposure on vascular function and endothelium- dependent relaxation.

In late sepsis, both nitric oxide- dependent relaxation responses and endothelial nitric oxide synthase levels were decreased in septic mice arteries.

Polyphenols from red wine increase endothelial NO production leading to endothelium- dependent relaxation in conditions such as hypertension, stroke or the metabolic syndrome.

In rabbit lungs, endothelium- dependent relaxation in isolated pulmonary arterial rings was decreased in the control group and normal in the PTX group.

Key findings: In early sepsis mouse arteries, endothelium- dependent relaxation decreased and phosphorylation of the inhibitory Thr495 site in endothelial nitric oxide synthase increased.

The contrast media caused concentration- dependent relaxation of both types of arteries; this effect was relatively insensitive to the agent used to precontract the vessels.

Obesity-related impairment of endothelium- dependent relaxation of the aorta was markedly attenuated by temporary periaortic fat removal whereas obesity-related enhancement of contractile performance was unaffected.

Clinically, endothelial function and dysfunction have been mostly evaluated by the assessment of endothelial dependent relaxation, for example in response to acetylcholine or increase inflow.