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1Results: Thirty-one of 50 patients displayed abnormal translation.
2Callosally-projecting neurons displayed abnormal orientation of the apical dendrite and increase in the basal dendritic length.
3Moreover, p63-null mice displayed abnormal epithelial development and germ-line human mutations were found to cause ectodermal dysplasia.
4Transgenic lines also displayed abnormal shoot apical meristems (SAMs) and retarded growth at the vegetative stage.
5At early postnatal days, brains displayed abnormal ventricular size and hemispheric asymmetry due to altered brain water homeostasis.
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