1 In patients with DM2, hyperglycemia and diabetes duration contribute to brain atrophy.
2 EDS was independently associated with DM2 diagnosis, suggesting possible primary CNS hypersomnia mechanisms.
3 Odds ratio were calculated for the presence or absence of MS and DM2.
4 We aimed to determine the frequency of sleep disturbances in DM2.
5 Conclusions: The results highlight BMI as a risk factor for both DM2 and depression.
6 Further studies utilizing objective sleep measures are needed to better characterize sleep comorbidities in DM2.
7 Our results provide additional evidence of a putative relationship between DM2 and idiopathic pulmonary fibrosis.
8 FCHL subjects showed insulin resistant adipose tissue lipid metabolism, in contrast to DM2 and controls.
9 Insulin mediated glucose uptake was impaired to the same extent in both FCHL and DM2.
10 Despite being a significant risk factor on univariate analysis DM2 was not significant in multivariate analysis.
11 The aim of this study was to prospectively investigate the bidirectional relationship between depression and DM2.
12 Conclusions: In patients with symptomatic arterial disease, DM2 has an added detrimental effect on the brain.
13 However, the role of elevated ALT levels in subjects with overt DM2 has yet not been explored.
14 Conclusions: The changes in DBP-PPAR-γ axis observed in mice with diabetes were also detected in patients with DM2.
15 However, therapeutic use of 4Dm2m was limited by its short in vivo half-life.
16 Thirty patients with DM2 and 43 controls responded to the survey.
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