We compared the effects of chemical cross-linking on the biological activities of glucans.
2
The alternative complement pathway was not activated by any of the cross-linked glucans.
3
Natural and chemically modified glucans had little or no activity.
4
It is comprised of glucans that form semicrystalline granules.
5
However, the observed changes were modest, and any long-lasting effects of yeast-derived β-glucans remain to be elucidated.
Ús de glucan en anglès
1
Results: β-glucan was barely detectable in serum of volunteers at all time-points.
2
This work has identified regions associated with glucan binding, toxin activity, and immunity.
3
They are released from starch granules by the glucan hydrolases alpha-amylase and isoamylase.
4
This is the first report on the isolation of a neutral beta-D-glucan from dates.
5
The physical basis for the function of SEX4 as a glucan phosphatase is currently unclear.
6
Instead we identify two additional glucan binding sites located within the core LSF2 phosphatase domain.
7
Our gene expression findings suggest that LLCs and macrophages are stimulated following β glucan exposure.
8
Consistent with this hypothesis, we found S. mutans strains defective in glucan production were more acid sensitive.
9
These results indicate that the key factors in the responsiveness to beta-glucan are GM-CSF production and dectin-1 expression.
10
A gas1 mutant with an expected defect in glucan elongation showed a large increase in the polydisperse fraction.
11
And 80 per cent contained traces of glucan, which is found in the cell walls of most fungi.
12
Levels of a starch called beta-glucan go up, making the sugar-water that gets fermented into beer too gloppy.
13
Subjects in the β-glucan group ingested β-glucan 1000 mg once daily for 7 days.
14
These results indicate that human PGN and 1,3-beta-D-glucan recognition proteins function as complement-activating lectins.
15
Echinocandins target fungal beta-1,3 glucan synthesis and are used clinically to treat invasive aspergillosis.
16
Global transcriptomics revealed proton accumulation triggers the up-regulation of genes encoding functions involved in acid tolerance response in a glucan-dependent manner.