1
Previously, we showed that additional perivenous hepatocytes express GLUT-1 in fasting animals.
2
Conclusions: Increased intracellular iron has dual effects on insulin sensitivity in hepatocytes.
3
Moreover, it enabled ultrastructural analysis of virions produced by primary human hepatocytes.
4
Background: Expression of miR-122 is highly specific to hepatocytes of the liver.
5
Methods: Primocultures of isolated hepatocytes or 7777 cells were stimulated with HGF.
1
Conclusion: Energy metabolism plays an important role in hepatocyte apoptosis during ALF.
2
However, intravenous administration of FasL induced lethal liver hemorrhages and hepatocyte apoptosis.
3
Background & aims: Oncogenes were activated in experimental models of hepatocyte regeneration.
4
Results: ASOR could only combine with hepatocyte and distribute in the liver.
5
Increased hepatocyte apoptosis has been observed in association with hepatitis C infection.
6
The role of TNF and Fas-ligand in hepatocyte mitosis also was assessed.
7
Seven hours after the drug administration, hepatocyte apoptosis and necrosis both started.
8
Apoptosis is likely an important pathway that culminates in hepatocyte cell death.
9
Conversely, inducing hepatocyte senescence promotes fat accumulation in vitro and in vivo.
10
On ultrathin liver sections, condensed chromatin of rat hepatocyte nuclei was studied.
11
These data support the suggestion that ethanol increases hepatocyte sensitivity to FAS-mediated damage.
12
Currently, it is difficult to assess rapidly hepatocyte function before transplantation.
13
If no hepatocyte injury is present on biopsy, therapy may not be needed.
14
These results suggest that HCV is replicating in the human hepatocyte PH5CH cells.
15
At present, the mechanism by which RA stimulates hepatocyte proliferation is largely unknown.
16
In conclusion, this study demonstrated that GA improved ethanol-induced hepatocyte necroptosis in vitro.
