Furthermore, we evaluated the expression profile of the candidate genes during humanadipocyte differentiation.
2
Conclusion: Our data demonstrate that macrophages modulate humanadipocyte energy metabolism via an activation-dependent paracrine mechanism.
3
The humanadipocyte-specific apM-1 gene encodes a secretory protein of the adipose tissue and seems to play a role in the pathogenesis of obesity.
4
These data have been verified in in vitro-differentiated primary humanadipocytes.
5
However, inflammatory factors released from activated macrophages induced a marked expression of procalcitonin and CGRP in co-cultured humanadipocytes.
6
In addition, humanadipocytes treated with either GSK126 or vehicle exhibited comparable rates of glucose-stimulated triglyceride accumulation and fatty acid uptake.
7
We aimed to investigate the transcriptome of LXR and the role of LXR in the regulation of glucose uptake in primary humanadipocytes.
8
The knowledge of the association between the MR, fibrosis and the effects of an MR antagonist (MRA) in humanadipocytes remains very limited.
9
In contrast, PM20D1 is one of the most strongly TZD-induced transcripts in humanadipocytes, although not in cells from all individuals.
10
Humanadipocyte precursor cells were cultured in conditioned media for 12 days to differentiate adipocytes, followed by 12 days of culture in media containing thrombopoietin.