Significats de human airway en anglès

Importance: Measles virus spreads rapidly and efficiently in human airway epithelial cells.

The regulation of ACE2 release was investigated in polarized human airway epithelia.

Background: The human airway is believed to be acidified in asthma.

Methods: Primary BSMC were isolated from human airway tissue of asthmatic and non-asthmatic patients.

We found that polarized human airway epithelia expressed abundant FR alpha on their apical surface.

Our results underline the regenerative potential of patient-derived human airway stem cells in lung tissue engineering.

The ARAF mutations were shown to transform immortalized human airway epithelial cells in a sorafenib-sensitive manner.

LGL1-conditioned medium increased migration of fetal rat primary lung epithelial cells and human airway epithelial cells.

Of particular interest, entry independent of FR alpha was observed in primary cultures of human airway epithelia.

Results: Gram-positive bacteria Staphylococcus aureus or Streptococcus pneumoniae, induced release of CXCL8 from human airway epithelial cells.

Using a model of well-differentiated human airway epithelia, we found that apical MMP9 significantly increases transepithelial conductance.

In addition, we sought to determine which immune cells transfer MeV infectivity to the human airway epithelium.

In human airway samples from two asthma cohorts, we observed altered expression patterns of multiple clock genes.

PM10 exposure primes human airway epithelial cells to subsequent models of cell damage and influenza A virus exposure.

The molecular identity of this calcium influx pathway in human airway smooth muscle (HASM) remains unclear.

Nectin-4 sustains measles virus entry and non-cytopathic lateral spread in well-differentiated primary human airway epithelial sheets infected basolaterally.