Significats de hypertonia en anglès

The decrease in FA correlated with decrease in area only in hypertonia.

Objectives: Spastic hypertonia is originally believed to cause contractures from clinical observations.

Participants: Ten stroke patients with hypertonia and 10 healthy subjects matched for age and gender.

Hyperekplexia is characterised by neonatal hypertonia and an exaggerated startle reflex in response to acoustic or tactile stimuli.

To explain why some kits become hypertonic, we hypothesized that white matter injury was responsible for the hypertonia.

In spite of the abnormal increase in antigravity muscle tone during PS postoperatively, hypertonia was not present during wakefulness.

Methods: To evaluate the effect of hypertonia on contracture development, the rats received botulinum toxin injections after spinal cord injuries.

Startle disease affects newborn children and involves an exaggerated startle response and muscle hypertonia in response to acoustic or tactile stimuli.

Thus, hypertonia is associated with white matter injury, and a population of hypertonia can be identified by magnetic resonance imaging variables.

Hyperekplexia or startle disease is characterized by an exaggerated startle response, evoked by tactile or auditory stimuli, producing hypertonia and apnea episodes.

Conclusion: Passive movements of the knee induced a decrease of spastic hypertonia in stroke patients through a combination of reflexive and mechanical factors.

Objectives: To quantify changes in spastic hypertonia during repeated passive isokinetic knee movements in stroke patients and to assess the role of muscle activity.

Conclusions: This animal study provides new evidence that hypertonia is not the sole cause rather is the partial contributor of contractures after spinal cord injuries.

Individuals with mutations in SLC6A5 present with hypertonia, an exaggerated startle response to tactile or acoustic stimuli, and life-threatening neonatal apnea episodes.

Hypertonia and postural deficits are observed in cerebral palsy and similar abnormalities are observed in postnatal rabbits after antenatal hypoxia-ischemia.

The decrease in FA correlated with decrease in area only in hypertonia.