The otic capsule of patients with otosclerosis contains premature bone with numerous cartilaginous remnants.
2
In addition, the endolymphatic domain often detaches from the rest of the otic epithelium during epithelial closure.
3
Other nearby otic structures were unaffected.
4
Some investigators have proposed that the pathogenesis of otosclerosis is related to these cartilaginous rests in the otic capsule.
5
We conclude that human-induced pluripotent cell lines that have been reprogrammed using nonintegrating mRNAs are capable to differentiate into otic cell types.
6
We provide evidence that Hedgehog signalling from ventral midline structures acts directly on the zebrafish otic vesicle to induce posterior otic identity.
7
We report the expression pattern and construction of a transgenic zebrafish line for a transcription factor involved in otic vesicle formation and skeletogenesis.
8
This line provides an essential tool for the further study of zebrafish otic vesicle formation and the development and regeneration of the skeleton.
9
In type 2, we were able to detect the medial two thirds of the structure, but we failed to see the whole otic capsule portion.
10
The otic capsule of patients with otosclerosis contains premature bone with numerous cartilaginous remnants.
11
In addition, the endolymphatic domain often detaches from the rest of the otic epithelium during epithelial closure.
12
Other nearby otic structures were unaffected.
13
Some investigators have proposed that the pathogenesis of otosclerosis is related to these cartilaginous rests in the otic capsule.
14
We conclude that human-induced pluripotent cell lines that have been reprogrammed using nonintegrating mRNAs are capable to differentiate into otic cell types.
15
We provide evidence that Hedgehog signalling from ventral midline structures acts directly on the zebrafish otic vesicle to induce posterior otic identity.
16
We report the expression pattern and construction of a transgenic zebrafish line for a transcription factor involved in otic vesicle formation and skeletogenesis.