Diffusion tensor imaging and proton density-weighted images were used to identify damage in the raphenuclei.
2
Limitations: We did not assess the damage to the ascending projection fibers from the raphenuclei.
3
We hypothesized that depressive disorder due to brainstem infarction is associated with damage to the raphenuclei.
4
Decreased metabolism also was evident in the anterior nuclear group of the thalamus, raphenuclei, and locus ceruleus.
5
Serotonin being a neurotransmitter synthesized in the raphenuclei of the brain could serve as an alternative biomarker.
6
The dorsal raphenuclei receive light input from the circadian visual system and indirect input from the biological clock nuclei.
7
The serotonergic raphenuclei of the midbrain are principal centers from which serotonin neurons project to innervate cortical and sub-cortical structures.
8
Serotonin (5-HT) neurons located in the raphenuclei modulate a wide range of behaviors by means of an expansive innervation pattern.
9
That the oxytocin receptor is expressed in the serotonergic raphenuclei suggests a mechanism by which the two neurotransmitters may cooperatively influence behavior.
10
We postulate that the phenomena observed following cerebellar lesions are the result of alteration in the control of serotonergic neurons of the pontine raphenuclei.