1
However, the PDS decomposition did not produce radicals or reactive oxygen species.
2
There is evidence that reactive oxygen species are involved in its pathogenesis.
3
Production of reactive oxygen species was also followed by measurement of chemiluminescence.
4
The antioxidant, trolox, decreased the formation of reactive oxygen species and apoptosis.
5
The tumor suppressor gene p53 is activated by reactive oxygen species-generating agents.
6
These effects were associated with reduction in reactive oxygen species production.
7
These results suggest that reactive oxygen species are major regulator of BCA-induced apoptosis.
8
Furthermore, chlorogenic acid reduced the level of reactive oxygen species in IPEC-J2 cells.
9
Emerging evidence indicates that reactive oxygen species are important regulators of vascular function.
10
Moreover, compound 5a generated reactive oxygen species to a considerable extent.
11
Electron transport chain blockade provides an effective method of attenuating reactive oxygen species.
12
Perhaps species other than reactive oxygen intermediates should be considered as therapeutic targets.
13
The damage could be mediated by the increased levels of reactive oxygen species.
14
Moreover, increased reactive oxygen species in the inflamed preodontoblasts were observed.
15
Rod-shaped CNPs demonstrated greater reactive oxygen species scavenging ability compared to cube-shaped CNPs.
16
Mitochondria produce reactive oxygen species that may contribute to vascular dysfunction.
