The question is, what's really behind the mechanism, whether beta-amyloid or tau?

However, the difference in the tau estimates did not reach statistical significance.

Results: Propofol strongly promoted hyperphosphorylation of tau at several AD-related phosphorylation sites.

Long strings of proteins called tau formed, as seen in Alzheimer's disease.

All these glial abnormalities showed tau-positive immunoreaction not found in control cases.