The best imaging approach for localizing ectopic ACTH-secreting tumors is not defined.

The rise in pressure was significant after 8 h of ACTH infusion.

The increase in cortisol levels was preceded by a corresponding ACTH increase.

The transport mechanism for precursor ACTH thus appears to recognize other prohormones.

ACTH insensitivity results from a group of rare autosomal recessive genetic defects.

Serum cortisol concentrations and ACTH stimulation tests were used to detect CIRCI.

In addition, laboratory examination showed elevated plasma ACTH and serum cortisol levels.

The responses to all doses of ET-1 were similar during ACTH infusion.

Plasma GH concentration was decreased but plasma cortisol and ACTH remained unaffected.

However, many patients with ACTH resistance lack an identifiable genetic aetiology.

Results: A greater decline in ACTH was observed after cortisol injection in PTSD.

Possible mechanism of elevated ACTH in this disorder was briefly discussed.

This tumour type frequently contains peptide hormones of the ACTH family.

Cortisol levels remained elevated and ACTH increased during the 13-day period.

The ACTH response to CRF attenuated by repeated administration of CRF.

ACTH did not further enhance the maximal effect of AII on c-fos expression.