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Hormone secreted by the posterior pituitary gland (trade name Pitressin) and also by nerve endings in the hypothalamus; affects blood pressure by stimulating capillary muscles and reduces urine flow by affecting reabsorption of water by kidney tubules.
AdhE- mutants of E. coli fail to ferment glucose and introduction of adhEMeta restored the growth of such mutants when grown under fermentative conditions.
2
The adh.2C2 and adh.6D4 clones thus provide an accessible system for defining mechanisms controlling developmental plasticity in early T-cell development.
3
However, cloned adhEMeta did not confer ethanol utilization ability to Escherichia coli or to Pseudomonas aeruginosa, even though it was transcribed in both these hosts.
4
Conclusion: We conclude that rare ADH variants are specific for alcohol dependence.
5
Twenty-four hours after LDN, mean ADH levels had returned to normal values.
6
The incidence of ADH seems to be lower than in Western countries.
7
These localization patterns were basically the same in water-loaded and ADH-treated animals.
8
ADH was diagnosed by strict application of Page's and lesion extent criteria.
9
Only one case of ADH could be detected by imprint cytology.
10
Furthermore, ADH was significantly lower in alcoholic than in non-alcoholic cirrhotics.
11
This study demonstrated that ADH class I gene is progesterone-responsive in the uterus.
12
There were no significant differences in ADH levels in the open donor nephrectomy group.
13
We previously reported associations of AD with seven alcohol dehydrogenase (ADH) genes.
14
The ADH gene cluster may harbor a causal variant(s) for alcohol dependence.
15
Functional analysis of the ADH-SREs demonstrated these sites to be essential for ADH transcription.
16
Alcohol dehydrogenase (ADH) class I gene was one of the candidate genes.