Meanings of alveolar type in English

The number of alveolar type II cells has increased twofold.

These results were confirmed using primary murine alveolar macrophages and primary alveolar type II cells.

Results: All solutions caused significant damage of rat alveolar type II cells at 20 hours.

Morphological results demonstrated a significant hyperplasia of alveolar type II epithelial cells 10 days after bleomycin injury.

In addition, MK protein expression in cultured human alveolar type 2-like cells treated with DEX and RA was also determined.

Studies with isolated alveolar type II cells showed that uptake of GSH-I14 was Na+ dependent and inhibited by GSH.

In the alveolar septum, the gold particles were enclosed in vesicles observed in the cytoplasm of alveolar type I epithelial cells.

Although both alveolar type I and type II cells express CFTR, it has previously not been known whether type I cells transport Cl-.

ATII cells are also closely involved in innate immunity within the lung and are stem cells that differentiate into alveolar type I cells.

Prior work has shown that transforming growth factor-β (TGF-β) can mediate transition of alveolar type II cells into mesenchymal cells in mice.

These data suggest that alveolar type II cells can acquire features of mesenchymal cells in IPF lungs and that TGF-β can mediate this process.

Conversely, these alveolar type II cell characteristics were reduced in MLE-15 cells cultured in conventional 2D static culture systems.

Incubation of primary cultures of human alveolar type 2 cells with methemoglobin resulted in a similar pattern of chemokine release and signaling pathway activation.

After perfusion of the isolated rat lung with GSH-I14, the conjugate accumulated primarily in the alveolar type II cell as observed by fluorescence microscopy.

In the lung, RBD-1 mRNA expression localized to the tracheal epithelium while RBD-2 was expressed in alveolar type II cells.

SP-B is synthesized in alveolar type II cells as a preproprotein and processed to the mature peptide by the cleavage of NH2- and COOH-terminal peptides.