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1 DNA analysis showed no abnormalities in the beta amyloid precursor protein gene.
2 Levels of cortical beta- amyloid precursor protein were not significantly affected by drug treatment.
3 In addition, beta- amyloid precursor protein staining indicated impaired axonal transport.
4 Mutations in genes such as those encoding amyloid precursor …
5 The β-site amyloid precursor protein-cleaving enzyme BACE1 is a prime drug target for Alzheimer disease.
6 The function of the amyloid precursor protein (APP) in brain health remains unclear.
7 A functional candidate gene in this region is the beta- amyloid precursor protein (APP) gene.
8 Spheroids demonstrated increased immunoreactivity to amyloid precursor protein and neurofilament indicating probable impairment of axonal transport.
9 These include the amyloid precursor protein and presenilins that are intimately involved in the mechanisms of AD.
10 The amyloidogenic processing of amyloid precursor protein was also reduced, indicating that TUDCA interferes with Aβ production.
11 However, deposits of human-sequence Aβ in amyloid precursor protein transgenic mice and non-human primates bind very little PIB.
12 Various lengths of Aβ peptide are generated by proteolytic cleavages of the amyloid precursor protein (APP).
13 Beta- amyloid precursor protein (betaAPP) deposits were detected within the neuronal cytoplasm from 4 months of age onward.
14 Human beta- amyloid precursor protein (APP) transgenic mice are commonly used to test potential therapeutics for Alzheimer's disease.
15 The amyloid precursor protein (APP) has garnered considerable attention due to its genetic links to Alzheimer's disease.
16 The amyloid precursor protein (APP) has been extensively studied because of its association with Alzheimer's disease (AD).
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This collocation consists of: Amyloid precursor through the time
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