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1
This suggests the altered local process involving MT in primary
axonal
degeneration
.
2
The complex molecular mechanisms that mediate secondary
axonal
degeneration
remain poorly understood.
3
We have addressed this question in a model system of vincristine-induced
axonal
degeneration
.
4
Molecules that promote
axonal
degeneration
could represent potential targets for therapy.
5
These changes were accompanied by cessation of TBI-induced chronic
axonal
degeneration
.
6
Knockdown of rtp also delays
axonal
degeneration
in severed olfactory axons.
7
Amyloid precursor protein immunoreactivity in the thalamus at 10 days suggested delayed
axonal
degeneration
.
8
Injury or genetically induced depletion of NMNAT2 triggers
axonal
degeneration
or defective axon growth.
9
This pattern of diffusion changes is similar to findings seen in immaturity or
axonal
degeneration
.
10
These data indicate N-APP is not the sole culprit in
axonal
degeneration
in adult nerves.
11
Selective synaptic and
axonal
degeneration
are critical aspects of both brain development and neurodegenerative disease.
12
The presented model thus mimics some of the aspects of
axonal
degeneration
in chronic progressive MS.
13
We found that transduction of Nmnat protein into severed axons via virus-like particles prevented
axonal
degeneration
.
14
The functional changes in the cavernous nerve anticipated and exceeded the
axonal
degeneration
detected by morphology.
15
Electromyographic findings and slow improvement of muscle weakness were compatible with
axonal
degeneration
of motor fibers.
16
The ImageJ thresholding analyses described here provide a descriptive method for quantifying the state of
axonal
degeneration
.
axonal
degeneration
axonal