A type of blood cell that helps prevent bleeding by causing blood clots to form.
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Indeed, HIV infection is often associated with increased levels of platelet activators.
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However, the molecular mechanism governing platelet aggregation is not yet fully understood.
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Results: Eleven of 156 patients had δ-SPD based on platelet ADP content.
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Conclusion: Hemorrheological parameters and platelet aggregation showed specific changes following carotid stenting.
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Conclusion: Hemorheological parameters and platelet aggregation showed specific changes following carotid stenting.
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Conclusions: Abnormal thrombocyte and hemoglobin levels at diagnosis were associated with poorer survival.
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The thrombocyte lineage appeared the most sensitive to irradiation.
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Our five patients were undergoing evaluation for thrombocyte when the bone marrow biopsy revealed the presence of mastocytosis.
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The total body Cl of ultrafilterable platinum correlated well with the creatinine Cl and the thrombocyte surviving fraction.
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We found increased thrombocyte GPX activity at the two highest dosage levels in women only, but not in men.
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ITP is an autoimmune disease which results in low blood platelet counts.
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The present investigation has studied the influence of A23187 on blood platelet aggregation and secretion.
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Heparin-induced thrombocytopenia refers to the decrease in blood platelet count following the administration of heparin, which is an anticoagulant.
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However, a low dose of cyclosporine A resulted in a sustained normal range of blood platelet count and negative proteinuria.
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Whole blood platelet aggregometry and clot signature analyzer parameters did not show any sign of activation of platelets or the coagulation system.
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Results: During treatment the leukocyte count decreased remarkably, and the patient developed lymphopenia together with a paradoxical increase in her blood platelet count.
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The low peripheral blood platelet count is caused by premature platelet destruction by self-reacting antibodies in addition to an impairment of platelet production.
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The discoid form of blood platelets is important to their function in hemostasis.
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Blood platelets are notoriously difficult to preserve in vitro for long periods of time.
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The hemophiliac, or H gene, produces male issue with a lack of blood platelets.
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One of these compounds is thromboxane, which encourages blood platelets to aggregate into clots.
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Photoreceptor rod cells and blood platelets are remarkably different, yet both illustrate a similar phenomenon.
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Both medicines help stimulate the production of blood platelets.
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Unstimulated blood platelets have a characteristic discoid form supported by a circumferential band of microtubules.
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Nanomolar concentrations of cerastocytin induce aggregation of blood platelets.
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Blood platelets and fibrinolytic activity may play an important role in the development of these complications.