1
Hierarchical cluster analysis of chromosomal imbalances revealed a closer relationship among cHL-NS than other subtypes.
2
There were no products of adjacent segregation, since the six chromosomal imbalances were unrelated to the translocation.
3
These tumors almost always possess unbalanced gain of the 17q, along with many additional recurrent chromosomal imbalances.
4
We profiled chromosomal imbalances in the different subtypes and investigated the relationships between clinical parameters and genomic aberrations.
5
By following this protocol, clinicians and researchers alike will be able to learn how to characterize their patients' chromosomal imbalances using DECIPHER.
6
In addition, multiple pregnancy losses and infantile deaths occurred in this family, suggesting that these chromosomal imbalances may produce a lethal phenotype.
7
The characteristic pattern of chromosomal imbalances distinct from other B-cell lymphomas suggests a specific pathway of genetic changes associated with this lymphoma.
8
In these tumors, the centrosome number was significantly higher than in a control tumor without a detected TP53 mutation and with few chromosomal imbalances.
9
We analyzed chromosomal imbalances in 39 tumor samples from primary male breast cancer by comparative genomic hybridization (CGH) and correlated CGH findings with clinicopathological factors.
10
Chromosomal imbalances were detected from average comparative genomic hybridization profiles for each entity.
11
Chromosomal imbalances were only detected by PCR if at least 20% of the cells carried the numerical aberration.
