1
It is suggested that the acute component of cutaneous hypersensitivity is mediated by Ig.
2
Delayed cutaneous hypersensitivity responses and Ab responses to T cell-dependent antigen were severely impaired.
3
Eleven of the 13 ADR were cutaneous hypersensitivity reactions, including one fatal case of Stevens-Johnson syndrome.
4
An excellent correlation was observed between delayed cutaneous hypersensitivity to DNCB and the clinical extent of malignancy.
5
Immunization resulted in the development of anti-antigen antibody and enhanced immediate cutaneous hypersensitivity to the 31-3B6 antigen.
6
Cutaneous hypersensitivity responses to house dust mites did not differ between treatments.
7
Cutaneous hypersensitivity responses to OVA in PR + Methyl progeny were intermediate to and not different from the responses of CON and PR sheep.