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1 Bone marrow- derived macrophages were also used to study changes mediated by hyperglycemia.
2 Repolarization effect of AS-IV in vitro on bone-marrow- derived macrophages was also detected.
3 Human postmortem lung tissue, human blood- derived macrophages , and pulmonary microvascular endothelial cells.
4 Monocyte- derived macrophages primarily fostered sprouting angiogenesis within the portal vein tract.
5 Angptl4 expression in peritoneal and bone marrow- derived macrophages was highly induced by lipids.
6 Celastrol capable of antagonizing inflammatory responses attenuated the resistin secretion from THP-1- derived macrophages .
7 The results with BAY were confirmed in primary human monocyte- derived macrophages and alveolar macrophages.
8 Human blood- derived macrophages and human pulmonary microvascular endothelial cells were infected with S. pneumoniae.
9 Tissue-resident macrophages can develop from circulating adult monocytes or from primitive yolk sac- derived macrophages .
10 In bone marrow- derived macrophages , foam cell formation was enhanced after inhibition of hh signalling.
11 When stimulated with FhTLM blood monocyte derived macrophages adopt an alternative or regulatory phenotype.
12 However, the regulation of CD36 expression in human monocyte- derived macrophages has not been fully elucidated.
13 Neither silica nor SPIO treatment elicited direct cytotoxic or pro-inflammatory effects in bone marrow- derived macrophages .
14 Atherosclerosis is a low-grade inflammatory disorder, with monocyte- derived macrophages as major drivers of plaque formation.
15 IFN-γ and LPS stimulation induces RGC32 expression in primary peritoneal macrophages and bone marrow- derived macrophages .
16 Many of the findings in vivo were recapitulated by hyperglycemia in mouse bone marrow- derived macrophages .
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