Meanings of embryonic lethality in English

The major phenotype observed in the progeny of injected animals was embryonic lethality.

There is no evidence of embryonic lethality and the mutant mice grow normally.

Homozygous deletion of Hic1 in mice results in major developmental defects and embryonic lethality.

Disruption of CCC assembly or function results in embryonic lethality.

Targeted disruption of the Fli1 gene results in embryonic lethality.

NuSAP deficiency in mice leads to early embryonic lethality.

Complete MPC2 deficiency resulted in embryonic lethality in mice.

Early embryonic lethality precludes analysis of the physiological requirements for Thoc1 in the developing embryo or adult.

Surprisingly, TF deficiency resulted in embryonic lethality due to abnormal extra-embryonic vessel development and defective vitelloembryonic circulation.

Complete depletion of H3.3 leads to developmental retardation and early embryonic lethality.

A null mutation of Hhex results in a failure to form the liver bud and embryonic lethality.

Genetic analysis of a T-DNA insertional mutant suggested that loss of function of ANK6 results in embryonic lethality.

Deletion of both genes results in embryonic lethality that can be rescued by transgenic expression of either receptor.

The complete knockout of TF in mice causes embryonic lethality associated with failure of the yolk sac vasculature.

However, the embryonic lethality of Myb-null mutations has been an impediment to identifying roles for c-Myb during lymphocyte development.

The endothelial cell-specific deletion of Gas in mice causes early embryonic lethality with massive hemorrhage and a disorganized vasculature.