However, no or a reduced G-protein regulation of uptake can be initiated.

However, the chimera with a second and third double-loop further enhanced G-protein activation.

Conclusion: Low expression of RGS2 contributes to increased G-protein-coupled signaling in hypertensive patients.

The actions of dopamine are mediated by a family of five G-protein-coupled receptors.

Chemokines classically mediate their effects by binding to G-protein-coupled receptors called chemokine receptors.

An additional less potent but superadditive inhibitory effect on the G-protein is suggested.

Palmitoylation is a reversible, post-translational modification observed in a number of G-protein-coupled receptors.

It contains unusually low levels of 48k-protein and very high levels of G-protein.

Features important for intracellular G-protein activation and signal transduction are revealed.

The best-known effects of 2-AG are mediated by G-protein-coupled cannabinoid receptors.

Apelin is a peptide known as the ligand of the G-protein-coupled receptor APJ.

Receptor-mediated modulation of ion channels generally involves G-proteins, phosphorylation, or both in combination.

These receptors couple to multiple G-proteins and regulate intracellular signaling pathways.

We report an impaired obstacle-avoiding response of roots in Arabidopsis lacking a heterotrimeric G-protein.

Neuronal G-protein-gated potassium (GIRK) channels mediate the inhibitory effects of many neurotransmitters.

G-protein-coupled receptors comprise the largest family of mammalian transmembrane receptors.