1
FKBP5 regulates the cortisol-binding affinity and nuclear translocation of the glucocorticoid receptor.
2
Binding of HNF-6 to DNA is required for inhibition of glucocorticoid receptor activity.
3
We provide evidence that flurandrenolide functions via the glucocorticoid receptor-mediated pathway.
4
These effects of HC were significantly reversed by the glucocorticoid receptor antagonist RU486.
5
They function through the glucocorticoid receptor, a member of the nuclear receptor superfamily.
6
The glucocorticoid receptor (GR) is a major drug target in inflammatory disease.
7
This region of BAF250 is capable of interacting directly with the glucocorticoid receptor in vitro.
8
CAR appears to be a primary glucocorticoid receptor-response gene.
9
The DEGs were enriched for various pathways, with glucocorticoid receptor signaling as the most significant.
10
This effect of dexamethasone could be reversed by coincubation with the glucocorticoid receptor antagonist RU 38486.
11
Down-regulation of the glucocorticoid receptor was considered as an instantaneous event or as a gradual steroid-receptor-mediated process.
12
As with other steroid receptors, the glucocorticoid receptor is associated with several other proteins in a heterocomplex.
13
Parallel cultures were supplemented with prednisolone with or without the glucocorticoid receptor antagonist mifepristone in various concentrations.
14
We find that chromosomal glucocorticoid receptor binding sites correlate with positive but not with negative local epigenomic effects.
15
The receptor is distinct from glucocorticoid receptor and CBG as determined by competition studies using dexamethasone and hydrocortisone.
16
The glucocorticoid receptor (GR) is a member of the steroid receptor family of ligand-activated transcription factors.
