Meanings of glucocorticoid therapy in English

The results suggest that THS was stable following treatment with glucocorticoid therapy.

Stage I disease should receive observation before glucocorticoid therapy.

There was no difficulty in clinical controls and no change in antihypertensive drugs related to glucocorticoid therapy.

Patients were treated immediately with glucocorticoid therapy.

We suggest examining the efficacy of ACTH in preventing human osteonecrosis, a devastating complication of glucocorticoid therapy.

Purpose: Osteoporosis and associated fractures are major concerns for patients with systemic rheumatic diseases on long-term glucocorticoid therapy.

In addition, discrete clinical predictors of incident vertebral fractures were evident early in the course of glucocorticoid therapy.

None of the NR3C1 haplotypes showed statistically significant association with glucocorticoid therapy success.

Results: Anti-IL-5 was well tolerated in all patients and lowered peripheral blood eosinophil counts despite ongoing systemic glucocorticoid therapy.

Conclusion: Attention to fracture risk assessment in patients undergoing glucocorticoid therapy and timely bisphosphonate treatment should lead to fewer fractures.

Our case supports an autoimmune pathophysiology for xanthomatous hypophysitis and suggests that high dose glucocorticoid therapy as a treatment option.

The evidence supporting glucocorticoid therapy for spinal cord injury is controversial; however methylprednisolone continues to be widely employed in this setting.

This form of asthma is often uncontrolled, marked by refractoriness to standard therapy, and shows persistent airway eosinophilia despite glucocorticoid therapy.

Antiresorptive therapy has been evaluated only in those premenopausal women who are on glucocorticoid therapy and in those with primary hyperparathyroidism.

Conclusions: TBS can be used as an early indicator of altered bone quality stemming from glucocorticoid therapy or, possibly, more severe asthma.

Second, they identify enzymes required for ceramide synthesis as therapeutic targets for combating insulin resistance caused by nutrient excess or glucocorticoid therapy.