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Developmental expression revealed that hsc4 transcripts are enriched in cells active in endocytosis and those undergoing rapid growth and changes in shape.
2
HESO was found to inhibit cell growth and induce apoptosis in HSC4 and HN22 oral cancer cells.
3
NC decreased cell viability in both HSC3 and HSC4 cell lines; further analysis demonstrated that cell viability was reduced via apoptosis.
1
Silymarin dose-dependently inhibited the proliferation of HSC-4 oral cancer cells and promoted caspase-dependent apoptosis.
2
Western blotting was used to evaluate the expression of the FANCD2 in HSC-4 cells.
3
TUNEL showed that the silencing of FANCD2 significantly increased apoptosis in HSC-4 cells induced by radiotherapy.
4
The present study showed that shRNA interference could effectively and stably silence FANCD2 expression in HSC-4 cells.
5
Intracellular oxidants such as hydroperoxides and hydrogen peroxides were scavenged in HSC-4 or HT-1080 cells by NHE water.