1
Recent studies revealed that several DC subsets are also infected with HTLV-1.
2
To standardize HTLV-1 qPCR, preparation of a well-defined reference material is needed.
3
Discussion: The clinical and epidemiological aspects of HTLV-1 infection are discussed.
4
Conclusion: HTLV-1 and its associated neurological disease has a marked impact on QoL.
5
Their removal strongly reduces the ability of HTLV-1-producing cells to infect target cells.
6
Thus, the number of HTLV-1 infected people in the world is probably much higher.
7
Many laboratories in Japan have developed different HTLV-1 qPCR methods.
8
HTLV-1 biofilm-like structures represent a major route for virus transmission from cell to cell.
9
We have expressed C-terminally-truncated forms of HTLV-1 protease with and without N-terminal His tags.
10
Cytotoxic T-lymphocytes are critical in the clearance of chronic viral infections such as HTLV-1.
11
We conclude that rheumatological disease is associated with HTLV-1 through geographical rather than aetiological means.
12
The addition of the N-terminal His tag dramatically decreased the activity of HTLV-1 protease forms.
13
HTLV-1 genotyping generates valuable molecular epidemiological data to better understand the evolutionary history of this virus.
14
This study provides robust data to support the development of cost-utility analysis of interventions for HTLV-1.
15
Moreover, no case of HTLV-1 seroconversion could be documented over time by studying such sequential samples.
16
HTLV-1 copy numbers obtained by these three methods were similar, suggesting that their results were accurate.