1
Alignment of sequence reads performed using bwa-mem against human b19 reference genome.
2
EBV and rIL-4 induce T cell-independent IgE production by normal human B cells.
3
Induction of IgE synthesis in human B cells requires two signals.
4
Conclusion: These findings indicate that SV40 can establish persistent infections in human B lymphocytes.
5
B-ALL cells were highly glycolytic and primary human B-ALL samples were dependent on glycolysis.
6
Human B lymphoid cells develop and proliferate in this model.
7
We confirmed dampened dissemination potential of human B lymphomas that overexpress GZMA and RASGRP1.
8
We noted somatic LKB1 deletion or mutation at low frequency in human B-cell lymphoma.
9
The early steps of differentiation of human B cells into plasma cells are poorly known.
10
Our results demonstrate that Btk is essential in regulating thresholds for human B cell tolerance.
11
BCL6 is the product of a proto-oncogene implicated in the pathogenesis of human B-cell lymphomas.
12
Genomic clones from the 380 cell line ( human B cell leukemia) were analyzed.
13
We studied the transduction of primary human B lymphocytes and myeloma cells with lentiviral vectors.
14
Here, we characterized human B cells expressing T-bet and analyzed their dynamics during viral infections.
15
MAbs were injected into hemophilia A mice, followed by injection of human B domain-deleted fVIII.
16
Most human B-cell non-Hodgkin's lymphomas arise from germinal centers.
