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Women with a past medical history of P-EC have a baseline hypercoagulable state postpregnancy.
2
Congenital and acquired hypercoagulable states arise from an imbalance between procoagulant and anticoagulant forces.
3
This includes a hypercoagulable state and the associated thromboembolism.
4
Thus, there is ongoing dispute about the impact of a hypercoagulable state upon progression of atherosclerosis.
5
Our findings suggest that a hypercoagulable state in heart failure can be modified by warfarin therapy.
6
Heart failure is associated with a hypercoagulable state.
7
Blood workup for preexisting hypercoagulable state was normal.
8
More recently, the hypercoagulable state which results from diminished response to APC has also been associated with arterial thrombosis.
9
Increased survival in recent years is consistent with improved management of hypercoagulable states as well as improved general care.
10
Thrombocytosis was shown to affect the blood hypercoagulable state and also have a negative prognostic value for GC patients.
11
Conclusions: An increased platelet aggregability contributes to the hypercoagulable states, that may increase the incidence of thrombosis in such patients.
12
The risk increases in patients with central lines and hypercoagulable states such as with malignancy, thrombophilia, or use of oral contraceptive pills.
13
The observed association likely does not reflect cause and effect, but rather that low serum albumin reflects a hyperinflammatory or hypercoagulable state.
14
A single-center, randomized, double-blind, placebo-controlled trial was performed to test the hypothesis that warfarin will modify a hypercoagulable state in heart failure.
15
It has been estimated that up to 10% of hypercoagulable state manifestations in patients with inflammatory bowel disease (IBD) are ischemic strokes.
16
We conclude from this pilot study that femorotibial PTA produces a hypercoagulable state which may result in failure of early patency due to rethrombosis.